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primary monoclonal antibody for human progesterone receptor (pr) ncl-pgr  (Novocastra)

 
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    Novocastra primary monoclonal antibody for human progesterone receptor (pr) ncl-pgr
    Primary Monoclonal Antibody For Human Progesterone Receptor (Pr) Ncl Pgr, supplied by Novocastra, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibody+ncl-pgr/primary+monoclonal+antibody+for+human+progesterone+receptor++pr++ncl+pgr/us08563599-705-23-28
    Average 90 stars, based on 1 article reviews
    primary monoclonal antibody for human progesterone receptor (pr) ncl-pgr - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    other:

    Article Title: Progestin receptor isoforms and prostaglandin dehydrogenase in the endometrium of women using a levonorgestrel-releasing intrauterine system.
    Article Snippet: The primary antibody raised in mouse against human progesterone receptor was used at a dilution of 1:40 (NCL-PGR, recognizing A 1 B subtypes, Novocastra Laboratories, Newcastle, UK).



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    Treatment effects on normal mammary gland epithelium. (a), whole mount images showing atrophic postmenopausal mammary gland epithelium with small lobular size in the control (con) and equol (EQ) groups compared to the estradiol (E2) group; arrowhead indicates a focus of cystic change; bars = 1.5 cm. (b), effects of E2 and EQ on epithelial density, measured by histomorphometry. (c), immunolabeling of normal epithelial cells for <t>estrogen</t> <t>receptor</t> <t>alpha</t> <t>(ESR1),</t> progesterone receptor (PGR), and the proliferation marker Ki67. Vertical bars indicate standard errors. * P < 0.05 and *** P < 0.001 vs. respective control group by Wilcoxon Rank Sum test.
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    Treatment effects on normal mammary gland epithelium. (a), whole mount images showing atrophic postmenopausal mammary gland epithelium with small lobular size in the control (con) and equol (EQ) groups compared to the estradiol (E2) group; arrowhead indicates a focus of cystic change; bars = 1.5 cm. (b), effects of E2 and EQ on epithelial density, measured by histomorphometry. (c), immunolabeling of normal epithelial cells for <t>estrogen</t> <t>receptor</t> <t>alpha</t> <t>(ESR1),</t> progesterone receptor (PGR), and the proliferation marker Ki67. Vertical bars indicate standard errors. * P < 0.05 and *** P < 0.001 vs. respective control group by Wilcoxon Rank Sum test.
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    primary monoclonal antibody for human progesterone receptor pr ncl-pgr  (Novocastra)
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    Treatment effects on normal mammary gland epithelium. (a), whole mount images showing atrophic postmenopausal mammary gland epithelium with small lobular size in the control (con) and equol (EQ) groups compared to the estradiol (E2) group; arrowhead indicates a focus of cystic change; bars = 1.5 cm. (b), effects of E2 and EQ on epithelial density, measured by histomorphometry. (c), immunolabeling of normal epithelial cells for <t>estrogen</t> <t>receptor</t> <t>alpha</t> <t>(ESR1),</t> progesterone receptor (PGR), and the proliferation marker Ki67. Vertical bars indicate standard errors. * P < 0.05 and *** P < 0.001 vs. respective control group by Wilcoxon Rank Sum test.
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    https://www.bioz.com/product/primary+antibody+ncl-pgr/primary+monoclonal+antibody+for+human+progesterone+receptor++pr++ncl+pgr/10__1158_slash_1078___0432__ccr___06___1426-124-21-26
    Average 90 stars, based on 1 article reviews
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    Image Search Results


    Treatment effects on normal mammary gland epithelium. (a), whole mount images showing atrophic postmenopausal mammary gland epithelium with small lobular size in the control (con) and equol (EQ) groups compared to the estradiol (E2) group; arrowhead indicates a focus of cystic change; bars = 1.5 cm. (b), effects of E2 and EQ on epithelial density, measured by histomorphometry. (c), immunolabeling of normal epithelial cells for estrogen receptor alpha (ESR1), progesterone receptor (PGR), and the proliferation marker Ki67. Vertical bars indicate standard errors. * P < 0.05 and *** P < 0.001 vs. respective control group by Wilcoxon Rank Sum test.

    Journal:

    Article Title: Estrogen effects on epithelial proliferation and benign proliferative lesions in the postmenopausal primate mammary gland

    doi: 10.1038/labinvest.2008.64

    Figure Lengend Snippet: Treatment effects on normal mammary gland epithelium. (a), whole mount images showing atrophic postmenopausal mammary gland epithelium with small lobular size in the control (con) and equol (EQ) groups compared to the estradiol (E2) group; arrowhead indicates a focus of cystic change; bars = 1.5 cm. (b), effects of E2 and EQ on epithelial density, measured by histomorphometry. (c), immunolabeling of normal epithelial cells for estrogen receptor alpha (ESR1), progesterone receptor (PGR), and the proliferation marker Ki67. Vertical bars indicate standard errors. * P < 0.05 and *** P < 0.001 vs. respective control group by Wilcoxon Rank Sum test.

    Article Snippet: Mammary gland sections were immunostained using commercially-available primary monoclonal antibodies for ESR1 (NCL-ER-6F11, Novocastra; Newcastle–upon–Tyne, UK), PGR (NCL-PGR, Novocastra), and Ki67 (Ki67/MIB1, Dako; Carpinteria, CA).

    Techniques: Control, Immunolabeling, Marker

    Immunolabeling of columnar cell lesions for estrogen receptor alpha (ESR1), progesterone receptor (PGR), and the proliferation marker Ki67. (a), labeling indices across treatments, stratified by lesion type. CCLs included columnar cell change (CCC) and columnar cell hyperplasia (CCH) lesions. Vertical bars indicate standard errors. * P < 0.05, ** P < 0.01, and *** P < 0.001 compared to respective normal epithelium values by Wilcoxon Rank Sum test. (b), representative immunostaining of normal duct (top row) and CCH (bottom row) for ESR1, PGR, and Ki67 in an E2-treated animal.

    Journal:

    Article Title: Estrogen effects on epithelial proliferation and benign proliferative lesions in the postmenopausal primate mammary gland

    doi: 10.1038/labinvest.2008.64

    Figure Lengend Snippet: Immunolabeling of columnar cell lesions for estrogen receptor alpha (ESR1), progesterone receptor (PGR), and the proliferation marker Ki67. (a), labeling indices across treatments, stratified by lesion type. CCLs included columnar cell change (CCC) and columnar cell hyperplasia (CCH) lesions. Vertical bars indicate standard errors. * P < 0.05, ** P < 0.01, and *** P < 0.001 compared to respective normal epithelium values by Wilcoxon Rank Sum test. (b), representative immunostaining of normal duct (top row) and CCH (bottom row) for ESR1, PGR, and Ki67 in an E2-treated animal.

    Article Snippet: Mammary gland sections were immunostained using commercially-available primary monoclonal antibodies for ESR1 (NCL-ER-6F11, Novocastra; Newcastle–upon–Tyne, UK), PGR (NCL-PGR, Novocastra), and Ki67 (Ki67/MIB1, Dako; Carpinteria, CA).

    Techniques: Immunolabeling, Marker, Labeling, Immunostaining

    Relative expression of genes related to estrogen receptor activity and proliferation in the mammary gland. (a), effects of estradiol (E2) and equol (EQ) treatment on expression of estrogen receptor alpha (ESR1), estrogen receptor beta (ESR2), progesterone receptor (PGR), trefoil factor 1 (TFF1), and Ki67 antigen (MKI67). (b), gene expression sorted by columnar cell lesion (CCL) status. All gene expression values were corrected for internal control gene expression and then expressed relative to control group values; lesion status effects were adjusted for any treatment group effects. * P < 0.05, ** P < 0.01, and *** P < 0.001 compared to respective control values.

    Journal:

    Article Title: Estrogen effects on epithelial proliferation and benign proliferative lesions in the postmenopausal primate mammary gland

    doi: 10.1038/labinvest.2008.64

    Figure Lengend Snippet: Relative expression of genes related to estrogen receptor activity and proliferation in the mammary gland. (a), effects of estradiol (E2) and equol (EQ) treatment on expression of estrogen receptor alpha (ESR1), estrogen receptor beta (ESR2), progesterone receptor (PGR), trefoil factor 1 (TFF1), and Ki67 antigen (MKI67). (b), gene expression sorted by columnar cell lesion (CCL) status. All gene expression values were corrected for internal control gene expression and then expressed relative to control group values; lesion status effects were adjusted for any treatment group effects. * P < 0.05, ** P < 0.01, and *** P < 0.001 compared to respective control values.

    Article Snippet: Mammary gland sections were immunostained using commercially-available primary monoclonal antibodies for ESR1 (NCL-ER-6F11, Novocastra; Newcastle–upon–Tyne, UK), PGR (NCL-PGR, Novocastra), and Ki67 (Ki67/MIB1, Dako; Carpinteria, CA).

    Techniques: Expressing, Activity Assay, Gene Expression, Control